Pathophysiology of Myocardial Infarction:
Pathophysiology of Myocardial Infarction – Myocardial infarction (MI a heart attack) focus on myocardium the muscles of heart and the changes that occur due to the sudden depreciation of circulation circulating blood by occlusion of plaque in carotid artery. The main changes is necrosis of myocardial tissue the word infraction come from latin ”infarcire” meaning to plug, it refer to the clotting of the artery.
Myocardial infarction is a disease that occur when blood supply to a part of other heart is interrupted which result in oxygen shortage cause Irreversible damage to the heart tissues it is usually a company can eat by accompanied by CBR chest pain atomic economic phenomenon such as looking pale setting feeling sick during the acute coronary occlusion blood flows into the coronary vessel the area of the muscle that has either 0 floor are related flow cannot sustain the cardiac activity lead to in cracked this process is called as myocardial infarction 17 years lead the muscle fibre used oxygen of stagnant blood cause the hemoglobin to become dark blue does infected area become Bruce Brown and the blood vessels of that area show lack of blood flow latest age divisible ball become highly permeable and fluid leak from the walls to the adjoining tissues make oedematous situation with the spelling of cardiac muscle cells due to reduction in silver metabolism between few hours blood supply stops and cell dies.
Cardiac muscles require about 1.3 ml of oxygen per 100 grams of muscle tissues permanent to remain alive this is in comparison to 8 ml of oxygen per 100 grams derived to normal resting left ventricular ventricle per minute if there is 15 to 30% of normal resting coronary blood flow which carries oxygen to the muscle the muscles will not die in the search certain portion of Lords in fact there is no Collateral blood flow lead to death of cardiac muscle.